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Case Study: Lateral Raphe Tear in a Competitive Athlete

Our Patient


Our patient is a 27 year-old male soccer player complaining of persistent low back pain, with difficulty maintaining engagement of the core muscles during high-intensity drills. The patient could not recall an acute injury, but he mentioned a sudden twisting maneuver during a game six weeks prior that “felt off,” although it caused no immediate pain.

The Challenge


Over the following weeks, the athlete noticed that activating his core was becoming progressively more difficult, especially during spriting and rotational actions. He also reported soreness in the lumbar spine and contralateral hip that worsened after training sessions, and posterior chain discomfort during running and cutting maneuvers.

Our Diagnostic Process


Clinical Examination

During the clinical exam, we noted poor co-contraction between the anterior abdominal wall and the paraspinal muscles. We also discovered altered breathing mechanics, with reduced lateral expansion.

Physical palpation revealed subtle densification along the lateral raphe region – a complex of dense connective tissue formed by the fusion of layers of the thoracolumbar fascia (TLF). The TLF is often described as a “fascial zipper” along both sides of the lower back whose dysfunction gives a sensation of “disconnect” rather than pain.

Our Treatment Protocol


Our treatment approach for this athlete was personalized and multimodal:

  • Prolotherapy injections targeting the lateral raphe to restore tensile integrity and improve fascial load-sharing.
  • DNS (Dynamic Neuromuscular Stabilization), a holistic method to retrain muscle activation in the abdominal canister, to restore reflexive co-contraction and normalize intra-abdominal pressure regulation.
  • Stecco Fascial Manipulation, an evidence-based method of releasing densifications in adjacent fascial planes to optimize force transmission and enhance frictionless gliding.

Results


After six weeks of multimodal treatment, the athlete reported improved abdominal wall activation, decreased compensatory lumbar pain, and restored efficiency of rotational action and sprint mechanics.

Discussion


Lateral raphe tears are common in athletes, often formed by repetitive microtraumas that worsen over time. In this case, we suspect that the raphe failed due to a sudden rotational load during soccer that may have created the initial tear, aggravated by TLF densifications that disrupted load transmission.

Raphe tears rarely present as localized pain at the site of injury. Instead, tensegrity of the abdominal canister is destabilized, and the athlete loses a reflexive co-contraction between the anterior and posterior stabilizers.

This results in:

  • Loss of connectivity during sport-specific movements
  • Difficulty activating the core muscles under load
  • Pain that manifest elsewhere due to compensatory strategies

The takeaway:

  • The presence or absence of pain can be misleading.
  • Lateral raphe dysfunction can masquerade as lumbar, hip, or pelvic pain.
  • For athletes struggling with “loss of abdominal connection” or unexplained weakness, the lateral raphe should be considered as a potential missing link.

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About the Author

Dr. Lev Kalika is a world-recognized expert in musculoskeletal ultrasonography, with 20+ years of clinical experience in advanced rehabilitative medicine. In addition to operating his clinical practice in Manhattan, he regularly publishes peer-reviewed research on ultrasound-guided therapies and procedures.

Dr. Kalika is an esteemed member of the International Society for Medical Shockwave Treatment ((SMST), and the only clinician in New York certified by the ISMST to perform extracorporeal shockwave therapy. He is also an active member of the American Institute of Ultrasound in Medicine (AIUM), and has developed his own unique approach to dynamic functional and fascial ultrasonography.

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In this instance, an athlete was originally diagnosed with minor quadriceps muscle strain and was treated for four weeks, with unsatisfactory results. When he came to our clinic, the muscle was not healing, and the patients’ muscle tissue had already begun to atrophy.

Upon examination using MSUS, we discovered that he had a full muscle thickness tear that had been overlooked by his previous provider. To mitigate damage and promote healing, surgery should have been performed immediately after the injury occurred. Because of misdiagnosis and inappropriate treatment, the patient now has permanent damage that cannot be corrected.

The most important advantage of Ultrasound over MRI imaging is its ability to zero in on the symptomatic region and obtain imaging, with active participation and feedback from the patient. Using dynamic MSUS, we can see what happens when patients contract their muscles, something that cannot be done with MRI. From a diagnostic perspective, this interaction is invaluable.

Dynamic ultrasonography examination demonstrating
the full thickness tear and already occurring muscle atrophy
due to misdiagnosis and not referring the patient
to proper diagnostic workup

Demonstration of how very small muscle defect is made and revealed
to be a complete tear with muscle contraction
under diagnostic sonography (not possible with MRI)

image

Complete tear of rectus femoris
with large hematoma (blood)

image

Separation of muscle ends due to tear elicited
on dynamic sonography examination

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