Improve Your Thoracic Mobility and Stability for Better Performance

March 25, 2025

Low back, neck and shoulder pain get a lot of attention in the healthcare world due to their frequent incidence, but very little mention is made of the thoracic spine. Yet thoracic mobility and stability play an important role in human movement, and dysfunction in the thoracic spine is a chief contributor to pain in other regions of the spine and body.

Learn about the thoracic spine, its role in sports and everyday movement, and how improving thoracic mobility can impact your overall health and physical performance.

Characteristics of the Thoracic Spine

The thoracic spine is made up of 12 vertebrae, numbered T1-T12, that bridge the cervical spine and the lumbar spine. The thoracic vertebrae are separated by flexible cartilage discs that provide cushioning and shock absorption, allowing for spinal rotation and flexion.The thoracic spine is the longest segment of the spinal column, and arguably the most important in terms of function.

Functions of the thoracic spine include:

  • Protects the spinal cord, lungs and heart
  • Provides structural support of the trunk
  • Helps to govern posture
  • Mediates load transfer between the upper and lower body during physical activity
  • Provides stability during rotational movement
  • Enables lateral flexion and rotation of the trunk
  • Houses nerves that control the muscles of the rib cage, lungs, diaphragm, and breathing apparatus
  • Provides a point of attachment for the costovertebral joints and muscles


The rib cage is connected to the thoracic spine by the costovertebral joints, attaching all but the bottom two ribs to the spine. The thoracic spine and rib cage work together to stabilize the chest and abdomen, provide a bony framework that protects the heart and lungs, and help to regulate breathing mechanics. A radiate ligament encases the costovertebral joints, providing stability and restricting rib cage movement to only slight gliding actions.

Trunk Movement in the Transverse (Rotational) Plane

The thoracic architecture is uniquely designed to enable trunk rotation, which is essential to all dynamic movement. Walking, running and most sports rely on trunk rotation to help maintain balance, transfer force loads, generate momentum, and enable arm and shoulder movement. Many sports like tennis, golf, baseball and hockey rely on trunk rotation to generate force when striking a ball or puck.

The thoracic spine serves as a key transitional point for distributing force loads between the head, shoulders and pelvis. It provides stability for rotational movements via its connection with the rib cage, and the surrounding muscles and fascia. When there is insufficient trunk rotation during dynamic movement, rotational stress is transferred to other structures such as the neck and shoulders, lower back, pelvis, hips, and lower extremities.

The thoracic spine serves as a key transitional point for distributing force loads between the head, shoulders and pelvis. It provides stability for rotational movements via its connection with the rib cage, and the surrounding muscles and fascia. When there is insufficient trunk rotation during dynamic movement, rotational stress is transferred to other structures such as the neck and shoulders, lower back, pelvis, hips, and lower extremities.

People who suffer from pain in the neck, shoulders and low back often have limited trunk rotation, and it can even cause issues like tennis elbow and golfer’s elbow. Reduced thoracic mobility can cause issues with the shoulder girdle, such as scapular dyskinesis and subacromial impingement.

Stiffness in the thoracic spine can limit your ability to breathe deeply by restricting movement of the lungs and diaphragm. Shallow breathing can have far-reaching repercussions for metabolic health, cellular function, tissue healing, and posture. Reduced oxygen flow can limit the ability of your cellular mitochondria to manufacture ATP, the energy molecule, resulting in fatigue and reduced physical performance.

Scapular Stability and Thoracic Mobility

Thoracic mobility is closely related to scapular stability – the ability of the muscles surrounding the scapula to hold the shoulder blade in position and control its movement. The shoulder blade plays a critical role in shoulder function, posture, and upper body movement. The loss of scapular stability is called scapular dyskinesis, where the scapula moves abnormally, especially when the arm is raised and lowered.

Good thoracic mobility enables proper scapular positioning and movement. At the same time, good scapular stability supports efficient thoracic mobility. When one or the other is deficient, it can affect shoulder function, posture, and upper body performance. In cases of scapular dyskinesis, the thoracic spine and ribs may compensate for excess shoulder and scapular movement by becoming stiff and immobile – a protective mechanism of the muscles

Good thoracic mobility enables proper scapular positioning and movement. At the same time, good scapular stability supports efficient thoracic mobility. When one or the other is deficient, it can affect shoulder function, posture, and upper body performance. In cases of scapular dyskinesis, the thoracic spine and ribs may compensate for excess shoulder and scapular movement by becoming stiff and immobile – a protective mechanism of the muscles

A stiff thoracic spine reduces the range of motion for overhead movements like reaching or throwing, forcing the scapula to overcompensate. This can increase stress on the rotator cuff or neck muscles and cause tension in the fascia, preventing the scapula from gliding smoothly along the rib cage. Strong scapular stabilizers enhance the thoracic spine’s ability to move by providing a solid base for dynamic actions.

The interdependent nature of the thoracic spine and scapula demonstrates how pain and dysfunction in one region of the body can cause loss of mobility in another area, disrupting the entire kinetic chain. The body’s biomechanical interdependence underscores the need for complex therapeutic approaches that are personalized and comprehensive.

Treating only the locus of pain without addressing other structures further along the kinetic chain can create additional compensatory changes that fail to improve functional mobility.

Thoracic Mobility and the Sympathetic Nervous System

The sympathetic nervous system (SNS) originates in the thoracic and lumbar regions of the spinal cord, extending from the first thoracic vertebra to the second or third lumbar vertebra, depending on individual anatomy.

A part of the autonomic nervous system, the sympathetic nervous system is often referred to as the “fight or flight” system, because it governs the body’s reactions to stressful or threatening scenarios. In such scenarios, the SNS releases norepinephrine from postganglionic neurons, sparking a cascade of physiological reactions.

Physiological changes triggered by the SNS include:

  • Release of epinephrine (adrenaline) into the bloodstream
  • Elevated heart rate, to pump adrenaline and oxygen throughout the body
  • Dilated pupils, to optimize vision
  • Bronchodilation, to enhance oxygen uptake
  • Blood vessel constriction and increased blood pressure
  • Inhibition of digestion, to redirect resources for immediate action
  • Increased perspiration

 

Mobility of the thoracic spine directly impacts the sympathetic nervous system due to the arrangement of nerve fibers of the sympathetic chain that run alongside the thoracic vertebrae. Restrictions in thoracic rotation can stimulate increased sympathetic activity, inducing a state of chronic stress and causing nerves to become hypersensitized. By contrast, optimal thoracic mobility can help to downregulate the SNS and promote a better balance between the sympathetic and parasympathetic nervous systems

Fascia and Thoracic Mobility

Tensegrity refers to a state of tensile integrity, where a system of individual components is held together under continuous elastic tension. In the human body, tensegrity is created by the myofascial system, where fascia’s elastic tension counterbalances muscle contraction and force production. Tensegrity holds vital organs in place during movement, creates symmetry, and enables the body’s systems to move and work in harmony.

Fascia is a type of connective tissue that encases and connects muscles and vital organs throughout the body. Fascia works together with muscles to control and guide movement, generate and transfer force loads, facilitate breathing, and provide a slippery ecosystem that enables nerves, blood vessels and other structures to glide without friction during movement.

Fascia is a type of connective tissue that encases and connects muscles and vital organs throughout the body. Fascia works together with muscles to control and guide movement, generate and transfer force loads, facilitate breathing, and provide a slippery ecosystem that enables nerves, blood vessels and other structures to glide without friction during movement.

The thoracolumbar fascia (TLF) consists of multiple layers of crosshatched collagen fibres, creating a girdling structure that separates the paraspinal muscles from the muscles of the posterior abdominal wall. The TLF contains nociceptive free nerve endings that are thought to contribute to low back pain.

When injured, the TLF can become densified and sticky, losing its elastic properties and reducing the ability of fascial layers to slide. Damaged fascia can cause reduced thoracic mobility, spasticity, and increased tension in the lower thoracic and lumbar spine. Nerve endings can become hyper-activated, triggering sensitization, local inflammation and pain, and inhibiting muscle action.

Thoracic Mobility, Breathing and the Pelvic Floor

Thoracic mobility promotes proper organ function, especially of the heart, lungs and breathing apparatus, but also of the pelvic organs. The relationship between the thoracic spine and pelvis merits attention, because improper breathing can cause pelvic organ prolapse if the pelvic bowl is not properly pressurized.

Intra abdominal pressure (IAP) is generated by the diaphragm as it flattens out to allow more air into the lungs. However, if the thoracic spine is tight, rib movement is restricted, making it difficult to fully inflate the lungs, and limiting diaphragm function.

When the thoracic spine curves forward, it increases resting pressure on the pelvic floor while reducing rib mobility.

Over time, poor posture from too much sitting can impact core and pelvic strength and stability. The transverse abdominis muscle of the deep core is directly connected to the thoracic fascia. Weakened muscles and limited thoracic mobility can cause various types of pelvic dysfunction, such as bladder incontinence, pelvic pain, constipation and diastasis recti during pregnancy.

The iliopsoas muscle, a powerful hip flexor that provides stability for the hip and lower back, originates at the twelfth vertebra of the thoracic spine. The hip flexors become tight with excessive sitting, creating poor posture and contributing to pelvic dysfunction. Because of the thoracic spine’s relationship to the pelvis, it is important to address thoracic mobility when treating pelvic floor disorders,

Back, Shoulder and Posture Issues

Poor thoracic rotation can affect the position and function of the shoulder blades, leading to scapular instability that affects posture and performance. To compensate for poor thoracic rotation, muscles of the neck, shoulders and back need to work harder. A stiff thoracic spine can cause shoulder impingement, and may contribute to rotator cuff pain.

Reduced thoracic mobility affects posture, causing stress on joints, muscles and fascia throughout the body. A forward head position, rounded shoulders and exaggerated low back arch are all associated with reduced thoracic mobility. When the thorax does not rotate well, the lumbar spine is forced to rotate, potentially causing lumbar disc compression and low back pain.

Factors that contribute to reduced thoracic mobility include:

  • Excessive sitting
  • Use of electronic devices
  • Poor breathing technique
  • Sedentary lifestyle
  • Seated sports like cycling or rowing

Thoracic mobility can be improved with exercises and stretches that rotate the middle spine through its full range of motion, and that flex the trunk laterally (bending to the side).

Optimize Your Thoracic Mobility in NYC

Movement efficiency is key to physical performance and pain-free mobility. A comprehensive biomechanical analysis can help to pinpoint inefficient movement patterns and identify their causes. At NYDNRehab, we use advanced technologies for data-driven results, enabling us to accurately assess movement parameters and measure patient progress. If you’re tired of feeling stiff and achy, contact us today, and let us help you get moving again!

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Resources

Erbes, Nicole A., et al. “Movement impairments in women with and without urinary urgency/frequency.” The Journal of Women’s & Pelvic Health Physical Therapy 45.4 (2021): 164-173.
Movement impairments in women with and without urinary urgency/frequency

Hunter, Donald J., et al. “Relationship between shoulder impingement syndrome and thoracic posture.” Physical therapy 100.4 (2020): 677-686.
Relationship between shoulder impingement syndrome and thoracic posture

Mellin, Guy. “Decreased joint and spinal mobility associated with low back pain in young adults.” Clinical Spine Surgery 3.3 (1990): 238-243
Decreased joint and spinal mobility associated with low back pain in young adults

Wang, Hao, et al. “Effects of trunk posture on cardiovascular and autonomic nervous systems: A pilot study.” Frontiers in Physiology 13 (2022): 1009806.
Effects of trunk posture on cardiovascular and autonomic nervous systems: A pilot study

Welbeck, A. N., et al. “Examining the link between thoracic rotation and scapular dyskinesis and shoulder pain amongst college swimmers.” Physical Therapy in Sport 40 (2019): 78-84.
Examining the link between thoracic rotation and scapular dyskinesis and shoulder pain amongst college swimmers

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About the Author

Dr. Lev Kalika is a world-recognized expert in musculoskeletal ultrasonography, with 20+ years of clinical experience in advanced rehabilitative medicine. In addition to operating his clinical practice in Manhattan, he regularly publishes peer-reviewed research on ultrasound-guided therapies and procedures.

Dr. Kalika is an esteemed member of the International Society for Medical Shockwave Treatment ((SMST), and the only clinician in New York certified by the ISMST to perform extracorporeal shockwave therapy. He is also an active member of the American Institute of Ultrasound in Medicine (AIUM), and has developed his own unique approach to dynamic functional and fascial ultrasonography.

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In this instance, an athlete was originally diagnosed with minor quadriceps muscle strain and was treated for four weeks, with unsatisfactory results. When he came to our clinic, the muscle was not healing, and the patients’ muscle tissue had already begun to atrophy.

Upon examination using MSUS, we discovered that he had a full muscle thickness tear that had been overlooked by his previous provider. To mitigate damage and promote healing, surgery should have been performed immediately after the injury occurred. Because of misdiagnosis and inappropriate treatment, the patient now has permanent damage that cannot be corrected.

The most important advantage of Ultrasound over MRI imaging is its ability to zero in on the symptomatic region and obtain imaging, with active participation and feedback from the patient. Using dynamic MSUS, we can see what happens when patients contract their muscles, something that cannot be done with MRI. From a diagnostic perspective, this interaction is invaluable.

Dynamic ultrasonography examination demonstrating
the full thickness tear and already occurring muscle atrophy
due to misdiagnosis and not referring the patient
to proper diagnostic workup

Demonstration of how very small muscle defect is made and revealed
to be a complete tear with muscle contraction
under diagnostic sonography (not possible with MRI)

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Complete tear of rectus femoris
with large hematoma (blood)

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Separation of muscle ends due to tear elicited
on dynamic sonography examination

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